Neuroinflammation in Stroke (häftad)
Format
Häftad (Paperback / softback)
Språk
Engelska
Antal sidor
224
Utgivningsdatum
2014-04-18
Upplaga
Softcover reprint of the original 1st ed. 2004
Förlag
Springer-Verlag Berlin and Heidelberg GmbH & Co. K
Medarbetare
Dirnagl, Ulrich (ed.), Elger, Bernd (ed.)
Illustratör/Fotograf
9 schwarz-weiße Tabellen 40 schwarz-weiße und 1 farbige Abbildungen Bibliographie
Illustrationer
1 Illustrations, color; 40 Illustrations, black and white; XII, 224 p. 41 illus., 1 illus. in color.
Dimensioner
210 x 148 x 13 mm
Vikt
291 g
Antal komponenter
1
Komponenter
1 Paperback / softback
ISBN
9783662054284

Neuroinflammation in Stroke

Häftad,  Engelska, 2014-04-18
1581
  • Skickas från oss inom 7-10 vardagar.
  • Fri frakt över 249 kr för privatkunder i Sverige.
Finns även som
Visa alla 1 format & utgåvor
Stroke is a major cause of death and disability in industrialized countries. To date, the medical need of efficient therapy for this de- vastating cerebrovascular disorder remains unmet. During the last decades, the development of pharmacological stroke therapies was aimed at improving patient outcome by restoration of cerebral blood flow or protection from acute neuronal cell death. Almost all of The participants of the workshop VI Preface these appraaches targeted the very early events after vascular occIu- sion. However, primarily for logistical reasons, only a small portion of strakes can be treated within 6-10 h after the insult. In recent years it has been recognized that strake pathophysiology is a dynamic pracess, and that delayed pracesses, which occur dur- ing the days and weeks following arterial occIusion, may lead to further deterioration or to impairment of recovery and rehabilitation in subacute and chranic stages. Evidence is accumulating that neu- rainflammation is a major player in these delayed pathophysiologi- cal pracesses. While some components of neurainflammation such as removal of cell debris and release of traphic factors may support recovery pracesses, others such as the generation of free radicals and other cytotoxic mediators are deleterious for brain tissue after isch- emia. Thus, neurainflammation after strake can be considered a dou- ble-edged sword, having potentially both detrimental and beneficial effects. As in other inflammatory reactions in the body, the cellular and humoral interactions are highly complex in the setting of neu- rainflammation.
Visa hela texten

Passar bra ihop

  1. Neuroinflammation in Stroke
  2. +
  3. The Menopause Brain

De som köpt den här boken har ofta också köpt The Menopause Brain av Dr Lisa Mosconi (häftad).

Köp båda 2 för 1806 kr

Kundrecensioner

Har du läst boken? Sätt ditt betyg »

Fler böcker av författarna

Innehållsförteckning

1 Matrix Metalloproteinases in Neuroinflammation and Cerebral Ischemia.- 2 Involvement of Tight Junctions During Transendothelial Migration of Mononuclear Cells in Experimental Autoimmune Encephalomyelitis.- 3 Cytokines in Stroke.- 4 Complement Activation: Beneficial and Detrimental Effects in the CNS.- 5 Inflammation in Stroke: The Good, the Bad, and the Unknown.- 6 Tetracycline Derivatives as Anti-inflammatory Agents and Potential Agents in Stroke Treatment.- 7 Induction of Mucosal Tolerance to E-Selectin Targets Immunomodulation to Activating Vessel Segments and Prevents Ischemic and Hemorrhagic Stroke.- 8 Protective Autoimmunity and Prospects for Therapeutic Vaccination Against Self-Perpetuating Neurodegeneration.- 9 Lessons from Stroke Trials Using Anti-inflammatory Approaches That Have Failed.- 10 Inflammation-Mediated Damage as a Potential Therapeutic Target in Acute Ischemic Stroke.- 11 Design Issues in Selected Recent or Ongoing Stroke Trials.- Previous Volumes Published in This Series.